Publications of Florian Wilfling
All genres
Journal Article (13)
1.
Journal Article
84 (10), pp. 1980 - 1994 (2024)
Autophagy preferentially degrades non-fibrillar polyQ aggregates. Molecular Cell 2.
Journal Article
26, pp. 378 - 392 (2024)
Combinatorial selective ER-phagy remodels the ER during neurogenesis. Nature Cell Biology 3.
Journal Article
120 (12), e2221712120 (2023)
In situ snapshots along a mammalian selective autophagy pathway. Proceedings of the National Academy of Sciences of the United States of America 4.
Journal Article
119 (39), e2209823119 (2022)
In situ structural analysis reveals membrane shape transitions during autophagosome formation. Proceedings of the National Academy of Sciences of the United States of America 5.
Journal Article
13 (1), 3041 (2022)
Cryo-EM structures of Gid12-bound GID E3 reveal steric blockade as a mechanism inhibiting substrate ubiquitylation. Nature Communications 6.
Journal Article
17 (10), pp. 3264 - 3265 (2021)
Autophagy ENDing unproductive phase-separated endocytic protein deposits. Autophagy 7.
Journal Article
Sample Preparation by 3D-Correlative Focused Ion Beam Milling for High-Resolution Cryo-Electron Tomography. Journal of Visualized Experiments (176), e62886 (2021)
8.
Journal Article
12 (1), 5364 (2021)
In situ cryo-electron tomography reveals gradient organization of ribosome biogenesis in intact nucleoli. Nature Communications 9.
Journal Article
586, pp. 796 - 800 (2020)
In-cell architecture of the nuclear pore and snapshots of its turnover. Nature 10.
Journal Article
9, e52459 (2020)
An advanced cell cycle tag toolbox reveals principles underlying temporal control of structure-selective nucleases. ELIFE 11.
Journal Article
22 (2), pp. 159 - 166 (2020)
Selective autophagy degrades nuclear pore complexes. NATURE CELL BIOLOGY 12.
Journal Article
80 (5), pp. 764 - 778.e7 (2020)
A Selective Autophagy Pathway for Phase-Separated Endocytic Protein Deposits. Molecular Cell 13.
Journal Article
10 (4), e0123341 (2015)
The Pro-Apoptotic BH3-Only Protein Bim Interacts with Components of the Translocase of the Outer Mitochondrial Membrane (TOM). PLOS ONE